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You can read here about bStats, what data is sent, and how to opt-out from sending statistics from your server. Since version 3.0, bStats is used to gather general usage statistics. On version 2.0 the NBTEditor started using MCStats for usage statistics. MCStats is now offline. To report bugs, on the latest version of the code, you can use the Issues Page at GitHub.ĭon't use the Issues Page to ask questions. Please check that you are using the most recent version of the plugin. Using the comments may help others that have the same problem.įor technical questions (errors/bugs) PM me directly. Please Read the FAQ before asking questions.įor questions about features and general help you can use the comments below or PM me. The awesome NBTEditor logo ( orig) was made by Sy_Gibbon from the french Minecraft community, they also have the documentation of NBTEditor and other plugins in french.Armor-Stand-Be-Gone (BoS to remove armor stands) by Taperpapers.Builds have not been approved by the BukkitDev staff. Arbitrary NBT modifications are not possible.ĭev. The variable system used by the NBTEditor ensures that only valid NBT changes are performed while providing a standard way to make those changes. Included is also an 'Item Storage' command to keep track of your Books of Souls and other edited items. ![]() No data files, no one-time customization, just keep the book safe and you can spawn as many entities as you like. Create bosses, custom villagers, flying creepers or anything you like. Conversion of tiles into '/setblock' commands Įdit now, spawn later: The Book of Souls is most powerful feature of the NBTEditor, it allows for easy entity customization.Tile customization (spawners, beacons, chests etc.).Conversion of items into '/give' commands.Customization of items (name, lore, unbreakable, modifiers, colors on leather armor etc.).Conversion entities into '/summon' commands.NBTUtil currently has a limited featureset compared to NBTExplorer.Windows Users NBTExplorer is a Windows. Robust system to edit of all entities and spawners using a book based UI (Book of Souls) The NBTExplorer zip and installer packages for Windows now include a second utility, NBTUtil.exe, for command-line control of NBT data. ![]() And with the help of the Book of Souls, it's possible to create highly complex entities and spawners with ease. Creating custom potions and tools with powerful enchantments is as easy as running a few commands. NBTEditor allows easy customization of many aspects of Minecraft, through the modification of NBT tags, on Spigot and CraftBukkit servers. ![]() And/or opting out of interest based ads ) looking adventure capitalist unblocked no flash attractive art and,! Adventure capitalist unblocked hacked dani's blog from y. ️ ️ ️□□□ ️ ️ ️ adventure capitalist hack is a collective computer game structured and distributed by the well known hyper hippo productions.
So, like other Pink Floyd or Waters endeavors, it's stretching the boundaries of what can be done, and more often than not it succeeded, in the wake of a triumphant fall of the real wall in Berlin. There is also an interesting, if not perfect, rendition of 'the Trial sequence' featuring a funny, scathing Tim Curry and judge played by Albert Finney. The Gerald Scarfe production design/animations are a wonder to behold for the fans, and there are a few other surprises as well. ![]() But that this is such an ambitious show, with such a huge, overpowering audience, is enough to suffice. Mitchell is good, the Band does a terrific job, and the Scorpions are hit or miss depending on how much of a fan you are. In fact, while he is usually singing the songs (there's a great bit when he's in part of the Wall right before, or after, the hotel-room smashing), the other acts sometimes steal the show, for better or worse. On top of this there is also the factor of the special guests, and even the whole spectacle itself, getting in the way of what Waters does best. It's the kind of live concert one wishes was experiences in person as opposed to on the screen like with the live show in other venues with the complete Pink Floyd, something is lost when experienced at home (the film is another matter, which is for my money one of the top 5 "expensive student films" ever made). ![]() On top of this is the complete stage-show of the Wall that made waves when first done in 1980. Here he is joined by such musicians as Bryan Adams, The Band (of Last Waltz 'Band' I mean), Cyndi Lauper, Joni Mitchell, Van Morrison, and the Scorpions among others. Live Roger Waters Format: Audio CD 432 ratings 1598 Get Fast, Free Shipping with Amazon Prime FREE Returns See all 7 formats and editions Streaming Unlimited MP3 14.99 Listen with our Free App Audio CD 15.98 22 Used from 6.45 16 New from 10.50 Includes FREE MP3 version of this album. So here, shortly after the fall of the Berlin Wall, Roger Waters brought forth a worldwide broadcast of the wall, complete with a spectacle for millions to experience, and of course not alone. It's more of a sensory experience than something traditional, like with the Who's rock operas. It's engaging (if a little obvious) with its messages on education, sex, the drainage of rock and roll, psychology, and the breakdown of society through fascism. It may not be Pink Floyd's best work, but on the form of ambition it could rightly rank with (or some would argue above) Dark Side of the Moon. ![]() The Wall is, indeed, one of the most awe-inspiring feats from the 70's rock scene. ![]() Our science team is put through the strictest vetting process in the health industry and we often reject applicants who have written articles for many of the largest health websites that are deemed trustworthy. Our team comprises of trained MDs, PhDs, pharmacists, qualified scientists, and certified health and wellness specialists.Īll of our content is written by scientists and people with a strong science background. We are dedicated to providing the most scientifically valid, unbiased, and comprehensive information on any given topic. We believe that the most accurate information is found directly in the scientific source. We examined the relationships between NLR, PLR, PNI, and overall survival and cancer survival using Cox-proportional hazard models.SelfHacked has the strictest sourcing guidelines in the health industry and we almost exclusively link to medically peer-reviewed studies, usually on PubMed. Subgroup analyses were done to examine the relation between hs-CRP categories and all-cause mortality and cancer mortality according to smoking status (current, former, and nonsmoker) and obesity status (BMI ≥25 kg/m 2, and <25 kg/m 2). We also conducted the same analysis on subjects excluding those who died within 2 years after medical check-ups. We used Cox-proportional hazard models after adjusting for potential confounders, such as age, sex, BMI, smoking status, hypertension, diabetes, total cholesterol level, high density lipoprotein (HDL)-cholesterol level, regular drinking (or not), regular exercise (or not), and monthly income. The HRs for all-cause and cancer mortality (including site-specific cancers) were estimated according to the 3 hs-CRP categories. The Kaplan–Meier method was used to describe the relationship between log-transformed hs-CRP and mortality from all causes and from cancer. Here, we sought to investigate the association between serum hs-CRP levels and mortality from all-causes, overall cancer, and site-specific cancer and we further examined the relationship of survival and cancer survival with inflammation-based prognostic scores including neutrophil lymphocyte ratio (NLR), platelet lymphocyte ratio (PLR), and prognostic nutrition index (PNI). However, no study has yet examined the possible association of hs-CRP levels and cancer death or site-specific cancer death in an Asian population. Recently, Lee and colleagues showed an association between hs-CRP concentrations and all-cancer risk and site-specific-cancer risks in 80,781 healthy Koreans ( 15). Some evidence suggest that there is a relationship between hs-CRP and cancer in Asian populations. For example, the 4 main causes of cancer mortality in the United States in 2007 were lung, breast, colorectal, and prostate cancer ( 13), whereas those in Korea in 2009 were lung, liver, stomach, and colon cancer ( 14). The common causes of cancer mortality among Koreans, however, appear to differ from those of people in other countries. Most of the studies on the influence of hs-CRP on cancer mortality have originated from western countries ( 7–12). Cancer Epidemiol Biomarkers Prev 21(11) 2076–86. Impact: As a marker for chronic inflammation, hs-CRP assists in the identification of subjects with an increased risk of cancer death. Elevated hs-CRP was also associated with an increased risk of site-specific mortality from lung cancer for sexes combined (2.53 ).Ĭonclusions: This study suggests that elevated levels of hs-CRP in apparently cancer-free individuals may be associated with increased mortality from all-causes and cancer, in particular, lung cancer in men, but not in women. The adjusted HRs (aHR 95% confidence interval ) of subjects with hs-CRP ≥3 mg/L for all-cause and cancer-related mortality were 1.38 (1.15–1.66) and 1.61 (1.25–2.07) in men, and 1.29 (0.94–1.77) and 1.24 (0.75–2.06) in women, respectively, compared with subjects with hs-CRP ≤1 mg/L. Results: During an average follow-up of 9.4 years, 1,054 deaths, including 506 cancer deaths, were recorded. Baseline serum hs-CRP levels were obtained and subjects were followed up for mortality from baseline examination until December 31, 2008. Methods: A total of 33,567 participants who underwent routine check-ups at a single tertiary hospital health-screening center between May 1995 and December 2006, and whose serum hs-CRP level data were available, were included in the study. We investigated the association between serum hs-CRP levels and all-cause mortality, cancer mortality, and site-specific cancer mortality in apparently cancer-free Koreans. Background: High-sensitivity C-reactive protein (hs-CRP) is an important inflammatory marker, and inflammation is known to be involved in the initiation and progression of cancer. ![]() In this test, Auto = Off all the time, since my Turbo Boost only enables at 65 degrees or below. Max Turbo: 95 to 100 degrees No Turbo: 70 to 80 degrees Auto Turbo: 70 to 80 degrees In this test, I just wanted to gauge the temperature range in Chrome while watching 4K YouTube video. Test 3 - Watching 4K video (Temperatures) While Auto was slower due to temps rising towards the end of the test, real world usage will hardly involve launching 7 apps consecutively, so I think Auto works well here. Split second loads are no problem for the MacBook Air because there isn't much time to heat up! So using Turbo Boost can really make a big difference. This test surprised me but also shows what Turbo Boost does best. Max Turbo: 32s No Turbo: 67s (!) Auto Turbo: 41s (Word, PowerPoint, Excel, OneNote, GarageBand, iMovie, Visual Studio Code) Then, I opened up 7 apps, each time waiting for the previous app to open before opening the next one. In this test, I restarted the computer and waited for background processes to settle. Test 2 - Opening 7 apps (App loading time, 7 apps is to reduce human error) (By no means am I saying that the thermal performance is good, because 1.1 GHz is a very low base clock to begin with.) ![]() Why was Auto cooler? I have no explanation.Īlso this is where I can say that the MacBook Air thermal throttles but doesn't overheat because it slows down its boost to stay below 100 degrees, but is perfectly capable of staying at or above base clock all the time. On Auto, Turbo Boost was never enabled as the temperatures were too high for the fans to slow down, thus explaining the almost identical score with No Turbo. Without Turbo, the Air completed the test at 1.1GHz all the way. Even on Max Turbo, the Air went at 3.2 GHz for half a second before running at 1.5 GHz the rest of the way. Let's get something straight, all three scores are dreadful. ![]() Max Turbo: 948 (const 100 deg) No Turbo: 645 (const 85 deg) Auto Turbo: 638 (const 80 deg) Test 1 - Cinebench R20 (Sustained workload) Macs Fan Control was set to activate the fan at 65 degrees and max out at 85 degrees, while Turbo Boost Switcher was set to disable at 3500 rpm fan speed and re-enable when fan speed dropped below 2800 rpm. I did this by (sigh) purchasing Macs Fan Control and Turbo Boost Switcher Pro. Therefore, I decided to try a middle ground - enable Turbo Boost, but disable it at the slightest hint of a sustained workload, and only re-enable it when temps are low again. But when I'm running at 50 degrees, Turbo Boost could definitely make for a snappier experience when opening an app or any other few-second tasks, and I'd be missing out on that with Turbo Boost permanently off. Never will the Mac go above 80 degrees (Cinebench aside). So, I decided to give Turbo Boost switcher a shot.ĭisabling Turbo Boost completely means that I never have to worry about temperatures at all. I love this beautiful gold MacBook Air, and boy do I want it to last. I live in a 32 degree climate so that doesn't help matters. I have definitely seen temperatures above 90 degrees in my workload, which is many apps and tabs but nothing you would consider Pro. 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First, we mapped the origins that become activated, using the DNA synthesis inhibitor hydroxyurea (HU) to inhibit fork progression and estimating the DNA content in the vicinity of fired origins by using whole genome DNA microarrays ( Heichinger et al., 2006) ( Fig. Since this pattern of DNA synthesis did not resemble a normal S phase, we asked whether normal S phase origins of replication were being used to replicate the genome and whether they fired in a coordinated fashion. Rather, it induces an extended period of continued DNA synthesis, resulting in approximately four doublings in DNA content. S1C) ( Kiang et al., 2009) as cells increased their DNA content from 2C to 32C, we found that cdc18 cdt1 co-oe does not show characteristics of repeated, periodic S phases that require G1–S gene expression. S1A,B) ( Sivakumar et al., 2004) or observing the G1–S gene expression marker Tos4-GFP ( supplementary material Fig. We analyzed the pattern of DNA synthesis and G1–S gene expression in the presence of excess Cdc18 and Cdt1. Therefore, we asked whether genome-wide coordination of origin firing is lost in cdc18 cdt1 co-oe, leading to local amplification, and if so, what features of replication origins might be responsible for that amplification. In these cells, the S phase controls, ensuring that an origin fires no more than once per round of replication, may be abrogated, because overexpression of a cdc18 phosphorylation-site mutant brings about some local amplification, particularly at the telomeres ( Mickle et al., 2007). In fission yeast, overexpression of the CDC6 homologue cdc18 in G2 induces re-initiation of DNA synthesis up to a DNA content of about 8C–16C ( Nishitani and Nurse, 1995) during co-overexpression with cdt1 ( cdc18 cdt1 co-oe), DNA content both increases more rapidly and attains a higher ploidy level (~32C) ( Gopalakrishnan et al., 2001 Nishitani et al., 2000 Yanow et al., 2001) (see supplementary material Table S1 for genotypes). ![]() Cdc6 and Cdt1 are components of the pre-replicative complex (Pre-RC) ( Bell and Dutta, 2002 Kelly et al., 1993) that bind at replication origins and recruit the mini chromosome maintenance (MCM) complex, the likely replicative helicase ( Bell and Dutta, 2002). Second, dysregulation of the replication factors Cdc6 and Cdt1 brings about increased DNA content in a range of organisms ( Arias and Walter, 2007 Gonzalez et al., 2006 Melixetian et al., 2004 Mihaylov et al., 2002 Vaziri et al., 2003). In fission yeast Schizosaccharomyces pombe this occurs by a largely normal S phase program given that normal S phase origins are used, periodic rounds of DNA synthesis are correlated with normal G1–S gene expression and cell mass doubling, and there is even replication of the genome ( Kiang et al., 2009). First, depletion of G2–M cyclin-dependent kinase (CDK) activity in yeast and fruit fly, and mouse and human cell lines leads to repeated DNA doublings without mitosis ( Arias and Walter, 2007 Kiang et al., 2009 Mihaylov et al., 2002). Unscheduled DNA synthesis can lead to local amplification ( Arias and Walter, 2007 Varshavsky, 1981) and genome instability ( Schimke et al., 1986 Seo et al., 2005 Varshavsky, 1981), and can be induced by two types of perturbation. We propose that these features predispose replication origins to re-fire within a single S phase, or to remain active after passive replication.įor stable genome inheritance, S phase occurs once per cell cycle, and within S phase the genome is completely and evenly replicated. Origins associated with amplification are highly AT-rich, fire efficiently and early during mitotic S phase, and are located in large intergenic regions. Specific origins are necessary for amplification but act only within a permissive chromosomal context. We find that S phase controls are attenuated and coordination of origin firing is lost, resulting in local amplification. To identify features of replication origins important for such amplification, we have investigated origin firing and local genome amplification in the presence of excess helicase loaders Cdc18 and Cdt1 in fission yeast. Failure in these controls can lead to local amplification, contributing to genome instability and the development of cancer. ![]() To ensure equal replication of the genome in every eukaryotic cell cycle, replication origins fire only once each S phase and do not fire after passive replication. It was released on November 22, 2011, and each DVD came with a green bandana as a bonus. The full series, including both the subbed and dubbed anime, the movie, and extras such as. Hetalia: The Beautiful World (Season 5) Todd Haberkorn 219 DVD 4 offers from 5. The English dubbed version made its premiere at the Anime Weekend Atlanta convention, taking place from September 30 to October 2, 2011. A description of tropes appearing in Hetalia: Axis Powers. At Otakon 2010, Funimation Entertainment announced the license of the film. Hetalia Axis Powers: Paint it, White! 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